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Recent molecular research have presented a lot of insights in the procedures that govern the development and progression of CLL, which include numerous novel mutated genes clustered in different functional pathways. The CLL epigenome is reprogrammed through the modulation of regulatory locations that appear de novo
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translocations or amplifications along with the genomic alterations currently existing in the original CLL, but lack the widespread mutations noticed in Principal DLBCL indicating which they may correspond to a distinct Organic classification.
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